Paresthesia of the lower extremities

Paresthesia of the lower extremities are one of the variants of the sensitivity disorder. This condition is characterized by sensations of tingling, crawling, numbness. Passing paresthesia of the extremities, as a rule, is caused by a direct mechanical type of irritation of the nerve, which is superficial. To such irritation carry blow or pressure. In addition, paresthesia of the lower extremities can arise due to temporary disorders in the blood supply. This, in turn, provokes a change in the conduct of nerve impulses. Such conditions occur often during sleep and when in an uncomfortable position.

Chronic paresthesias of the lower limbs are often symptoms of lesions of different areas of the nervous system. These include disorders of the primary type: tumor and infectious lesions, autoimmune and neurodegenerative processes, and others. Conditions can manifest themselves against the background of secondary lesions and may be complications of existing diseases. Thus, paresthesias of the lower extremities are observed in alcoholic polyneuropathy, deficiency of individual vitamins, metabolic disturbances (in diabetes mellitus, for example), atherosclerosis.

One of the known forms of the condition is considered to be a long-lasting numbness of the tongue, lips or chin.

The defeat of bundles of nerve fibers occurs with radiculitis. This disease is considered to be the most common lesions of the nervous peripheral system. The defeat of the spinal roots with radiculitis is a consequence of the disease of the spine (osteochondrosis). In this case, the elasticity of the intervertebral cartilaginous discs decreases, which results in the squeezing of the roots. As a result, there are feelings of numbness, pain.

Paresthesia can relate to the structure of vegetative-vascular paroxysm. As a rule, it occurs in the form of headache or heart pain, redness of the face, palpitations. At the same time, blood pressure and temperature increase, heart rate increases, chills begin. In some cases, the patient experiences unreasonable fear. Others are characterized by general weakness, darkening in the eyes, nausea, dizziness, sweating, as well as a decrease in blood pressure and pulse rate. Seizures can last from a few minutes to three hours. In many cases, they go without therapy.

Aggravation of vegetative-vascular dystonia is accompanied by cooling of the extremities. They also become wet and purple-cyanotic. Paresthesias in hands are manifested by numbness in the fingers, a feeling of crawling, a tingling sensation. In some cases, the patient feels pain. In this case, the sensitivity to cold effects increases significantly, the limbs look very pale. After an attack for several days, the patient may have a general malaise and a sense of weakness.

Persistent long-term paresthesias can be caused by ischemic lesions in the cortical representation of sensitivity or talcocorp afferent pathways. The states of limb numbness can develop both against the background of transistor ischemic attacks, and during an associated migraine.

Paresthesias may not have clear boundaries and have a burning nature. In most cases, such conditions occur due to focal lesions of the spinal cord or thalamus. Characteristic are the paresthesias of the angle of the mouth and the hand, arising from the lesions of the parietal lobe or thalamus. In addition, the conditions can develop sharply against the background of hemorrhage into the areas of the medial loop.

The main diagnostic sign include antigyokeratomes in the navel, pubis and knees from dark red to brown with a bluish tinge.